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Although energy metabolism status can also regulate intracellular ROS levels (e.g., glucose deprivation leads to a marked increase in cellular ROS) [53,54,55,56], our study demonstrated that supplementation with energy substrates such as sodium pyruvate in GGCT-knockdown cell models did not induce significant alterations in intracellular ROS levels

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In glutamate-exposed HT22 cells, -Oryzanol improved m , alleviated GSH depletion, and reduced the expression level of CAMKII, thereby protecting neurons from glutamate excitotoxicity [187]